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yxfbbiedtj · Wysłany: Pią 10:06, 04 Mar 2011

Blood of patients with asthma,[link widoczny dla zalogowanych], nitric oxide and endothelin -1 levels were measured in the clinical study

T increased. ET can activate 15 for a lipoxygenase and arachidonic ene pupil. Cause bronchoconstriction, chemotaxis of inflammatory cells and airway inflammation in the lung increased. {Blink inflammatory reaction and swelling Xin proinflammatory Cave necrosis factor, interleukin-1 boat and other cytokines prime the fork to increase ET synthesis and release. The formation of endothelin {wink vicious circle between cytokines. . {If this experiment. Library asthma attack to ease the most famous of elevated ET Ke. Suggest that ET is increased. The result of a very sexual cycle. Reduce the ET level or antagonistic E'I 'technology should approach the treatment of asthma indicate a new way to steep. N () in relaxing blood vessels, nerve transmission and regulation of immune function of information plays an important role in the fall of N0 by NO synthase (N (scorpion) catalyzed.. Asthma is a variety of inflammatory cells involved chronic inflammation. asthma attack the wall. macrophages. mast cells, neutrophils,[link widoczny dla zalogowanych], called on Pao luxuriant in tumor necrosis factor,[link widoczny dla zalogowanych], interleukin 1 and other elements under the action of the boat breaking Che live. the release of NO synthase increased. produce large amounts NO. NO addition to relaxation outside the bronchial smooth muscle. showed more cytotoxicity Jing t pay attention capillary oozing of blood after an increase in bone, DNA deamination,[link widoczny dla zalogowanych], bronchial epithelial cells and functional variability off screen. so that asthma symptoms get worse,[link widoczny dla zalogowanych]!. Bong results showed. asthma attack of N () and ET-1 was significantly higher than that of asthma and the control group steep remission (P <0.O1). Library asthma remission N () and ET-1 was still significantly higher baby (P <0.Ol or P <0.05) o and the library reported animal experiments similar to asthma. A = Because asthma is a chronic airway inflammation. airway epithelial cell damage. the release of ET and NO. Therefore, we believe that chronic inflammation is the library by air asthma exacerbation and remission and ET No one important reason for increased】 No features from consideration, or not smoking should be carefully selected outside of cochlear 【N () method to treat asthma. hormones not only Chong by-step ET · l from the release of airway epithelial cells. also selectively inhibit the N () sets into enzymes. alkali generation of less N0 , which prompted steep bank still needs to maintain a longer remission period of time of hormone treatment center study also revealed that asthma attack Wall elevated N () and ET, 1 is the book between the negative correlation. indicate that N () and ET-l The negative feedback regulation mechanism of smell. NO and ET-l is a pair of mutually antagonistic important factor in adjusting claims. Determination of NO and ET-l to r solutions of the severity of asthma. in asthma diagnosis, treatment and prevention play an important role in guiding.