nike sale After antioxidant treatment of corneal u

43nike9485qt · Wysłany: Wto 11:12, 15 Mar 2011

After antioxidant treatment of corneal ulcers change of energetic materials

There are also some special points , namely,[link widoczny dla zalogowanych], glucose and glycogen as its main metabolite, the glycolysis , pentose bypass aerobic oxidation and metabolism of three ways , and pentose -pass metabolism of epithelial cells is very active . Studies have shown that . . ~ 935 metabolism of glucose through this path that the repair of corneal epithelium ,[link widoczny dla zalogowanych], update soon related. The aerobic oxidation and endothelial cell metabolism of pentose shunt is less corneal epithelial and endothelial cells in many of the features are energy-consuming ,[link widoczny dla zalogowanych], require adequate energy supply . Study pointed out. Corneal epithelial injury . The glycogen reserves will fall significantly. Mild alkali burns , the corneal tissue ATP content also decreased significantly. . One. This study shows that untreated control group, the ATP was significantly lower than normal group , ADP also tended to decline , AMP was significantly increased ,[link widoczny dla zalogowanych], ATP / ADP ratio was significantly lower . This is a large number of corneal ulcers after the decomposition of tissue damage , cell death and compensatory energy-consuming activities related to the strength of cell . And by the antioxidant treatment, ATP treatment groups , though still below normal. But were significantly higher . And the SOD + CAT treatment group recovered the best, followed by tea polyphenols group . Vitamin C, vitamin E treatment group had the lowest . SOD + CAT treatment group and tea polyphenols in the treatment group also reached the normal level of ADP ; ATP / ADP ratio of the treatment groups have different degrees of recovery. High degree of material recovery with the performance of the same antioxidant treatment group (data are reported separately .) Previous studies have shown that oxygen free radicals in corneal tissue damage is the pathological one of the factors , but also in vivo activation of latent collagenase enzymes - Reduces lipid peroxidation damage, also reduces the activity of collagenase production and damage effects , thereby reducing the decomposition of corneal damage . the organization gradual recovery of energetic materials . while the improvement of high-energy material , but also conducive to the organization The results of further repair . another example of that antioxidant therapy is an effective means of treating corneal ulcers of 4
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