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yxfbbiedtj · Wysłany: Pią 4:12, 04 Mar 2011

Cerebral Infarction with serum bilirubin, Lipids of

7 / 33), 7.41% (2 / 33), 18.5% (5 / 33), twenty-two no statistically significant difference (P> 0.05). 3 discussions found in recent years, serum bilirubin can prevent lipoprotein and lipid oxidation, thereby reducing the formation of atherosclerotic plaques role of J. Atherosclerosis is the pathological basis of the importance of cerebral infarction,ghd piastre, which indicated that between serum bilirubin levels and cerebral infarction may be related. Data confirmed that patients with cerebral infarction compared with normal serum bilirubin levels were decreased ¨ J. This observation consistent with the above report, and further found that thrombotic cerebral infarction patients with lacunar infarction compared with serum bilirubin levels were significantly lower, indicating that low serum bilirubin,UGG stivali, indirect bilirubin in particular thrombotic cerebral infarction as risk predictor, but the index and lacunar infarction was no significant correlation. As the incidence of cerebral thrombosis and lacunar infarction different mechanisms, the former more by the brain caused by atherosclerosis, which causes the brain due to high blood pressure caused by small atherosclerosis, and low serum bilirubin leading to lipoprotein and lipid accelerated oxidation factors, and is therefore closely related to atherosclerosis. Hyperlipidemia is an important risk factor for cerebral infarction, but the promise of Clinical Emergency Medicine Volume 8, 2007, over 2 clinical study of the conclusions vary widely J, in addition to possible disease and hyperlipidemia between the onset of cerebral infarction there is a longer time period, follow-up time for different reasons, but also may be the genetic quality of the different study population,tory burch reva, groups in which regional differences, by researchers and exercise habits inconsistent hobbies, various risk factors influence the individual's share proportion of size. This study showed: TC, TG in the CI group and control group showed no significant difference, in addition to the above factors, this may lead to cerebral thrombosis occurred because of high blood lipid risk factors, not only to quantify the level of lipids, but also may related to changes in the nature of lipids, namely, the brain lipid oxidation may be a major atherosclerosis risk factors, and serum bilirubin levels reflect body lipid oxidation is an important indicator of the state. Lp (a) is a special kind of plasma lipoprotein levels in individuals fairly stable, mainly by genetic factors, almost regardless of age, sex, drugs, diet and general effect. So far, on the high Lp (a) the mechanism of cerebral infarction caused by the following two aspects that may be related to]: ① atherogenic role (in foam cell formation); ② promotion of thrombosis, and coagulation and fibrinolytic system-related, and epithelial cell adhesion molecules related. In conclusion,mulberry bags, bilirubin (TBIL, IBIL) decreased, Lp (a) levels and thrombotic cerebral infarction,mbt scarpe outlet, the development of a certain correlation to predict an important indicator of risk of cerebral infarction. This detection of a number of commonly used clinical indicators of stroke prevention and treatment monitoring can be used as a reference.